By L. Michael Prisant
A complete evaluate of all elements of high blood pressure within the aged utilizing the most up-tp-date medical information. subject matters variety from simple innovations, epidemiology and trials, and overview and administration, to pharmacologic remedy, distinctive populations, and adherence, all awarded with an emphasis at the optimum administration of sufferers. The authors research intimately the mechanisms of high blood pressure within the aged, the life-style trials and results trials that have been performed in older people, in addition to the issues of medical assessment, secondary high blood pressure, adherence, and objective organ harm. large discussions of pharmacologic treatment aspect the position of all of the significant drug sessions.
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Extra info for Hypertension in the Elderly (Clinical Hypertension and Vascular Diseases)
Aging is associated with endothelial dysfunction in healthy men years before the age-related decline in women. J Am Coll Cardiol 1994;24:471–476. 22. van der Loo B, Labugger R, Skepper JN, et al. Enhanced peroxynitrite formation is associated with vascular aging. J Exp Med 2000;192:1731–1744. 23. Chen J Brodsky SV, Goligorsky DM, Hampel DJ, Li H, Gross SS, Goligorsky MS. Glycated collagen I induces premature senescence-like phenotypic changes in endothelial cells. Circ Res 2002;90:1290–1298. 24.
Ageing Res Rev 2003;2:169–90. 14. Lakatta EG. Cardiovascular regulatory mechanisms in advanced age. Physiol Rev 1993;73:413–467. 15. Matz RL, Schott C, Stoclet JC, Andriantsitohaina R. Age-related endothelial dysfunction with respect to nitric oxide, endothelium-derived hyperpolarizing factor and cyclooxygenase products. Physiol Res 2000;49:11–18. 16. Lundberg MS, Crow MT. Age-related changes in the signaling and function of vascular smooth muscle cells. Exp Gerontol 1999;34:549–557. 17. Homma S, Hirose N, Ishida H, Ishii T, Araki G.
There remains a role for excessive vasoconstriction in the syndrome of hypertension, however, because systemic vasoconstriction and increased SVR contribute to both systolic and diastolic hypertension (Fig. 6). Overall, increased SBP can be the result of increases in stroke volume, arterial stiffness, or SVR, whereas DBP is decreased when central arterial stiffness increases. DBP thus varies directly with SVR and inversely with central arterial stiffness. The ability of increased SVR to cause increases in either SBP or DBP (depending on the degree of central arterial stiffness) causes otherwise unexpected differences in the therapeutic responses of SBP and DBP to Chapter 3 / Mechanisms of Hypertension 31 Fig.
Hypertension in the Elderly (Clinical Hypertension and Vascular Diseases) by L. Michael Prisant