By Anitha Varghese MBBS BSc MRCP, Dudley J. Pennell MD FRCP FACC

ISBN-10: 0443103011

ISBN-13: 9780443103018

This identify presents an simply digestible and transportable synopsis of the strategy on the way to go well with the desires of cardiologists and cardiothoracic surgeons wishing to acquaint themselves with what CMR can do, and what it can't. starting with an summary of a few of the elemental ideas of MRI, the subsequent chapters pay attention to the cardiac facet of CMR with a later part on its extra tested vascular makes use of.

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Extra resources for Cardiovascular Magnetic Resonance Made Easy

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9) and eosinophilic heart disease causing endomyocardial fibrosis. Cardiac sarcoid can also cause fibrosis, visible by LGE, often with dense punchedout lesions. Myocardial oedema may be present on T2W images in association with active inflammation. 13). RV non-compaction accompanies LVNC in a significant proportion of patients. Clinical manifestations include heart failure, ventricular arrhythmias and systemic embolic events. TTE and CMR are the imaging methods of choice with the criteria for diagnosis being a ratio of non-compacted to compacted myocardial layers of greater than 2.

Indications CMR can: 1. Determine LV and RV cardiac morphology and function normalized to sex, age and body surface area; 2. 13 (a) Four-chamber, (b) two-chamber and (c) mid-ventricular short-axis SSFP cine CMR images from a 68-year-old man with known LVNC highlighting the pathognomonic combination of multiple prominent ventricular trabeculations and deep intertrabecular recesses in communication with the ventricular cavity (black arrows). 3. 4. 5. 6. Detect myocardial oedema/iron/fat; Visualize myocardial fibrosis and protein deposition; Differentiate between DCM and cardiac dilatation due to CAD; and Distinguish between the cardiomyopathies (assign the phenotype).

MVO occurs in the first hours post-MI, predicts an unfavourable prognosis, and corresponds to areas of no-reflow. 4). 5). 4 Mid-ventricular short-axis view of an EGE study in an acute lateral MI showing a small dark zone of MVO embedded within the infarcted area (white arrow). 5 (a) Four- and (b) two-chamber views from a 74-year-old man who had a history of MI in the left anterior descending artery (LAD) territory. EGE shows an LV apical thrombus (black arrows) overlying a transmural chronic infarct.

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Cardiovascular Magnetic Resonance Made Easy by Anitha Varghese MBBS BSc MRCP, Dudley J. Pennell MD FRCP FACC


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